Author

L. V. Schnabel

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Assay:flow cytometry· 1 Organism:mouse· 1

Frequent Co-Authors

A. K. Berglund· 1 shared A. L. Hinson· 1 shared B. O. Diekman· 1 shared D. Howe· 1 shared E. H. Griffith· 1 shared G. A. Sessions· 1 shared J. D. Thompson· 1 shared J. M. Gilbertie· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperImmunology

A. K. Berglund

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3 papers

Recent·Most cited

TGF-β downregulates antigen processing and presentation genes and MHC I surface expression through a Smad3-dependent mechanism.

A. K. Berglund, A. L. Hinson, L. V. Schnabel

This study identifies that TGF-β downregulates MHC I surface expression and antigen processing genes via a Smad3-dependent mechanism in mesenchymal stem cells.

bioRxiv · Jan 30, 2023 · PreprintImmunology

Degenerative changes are associated with severity of anterior cruciate ligament injury within the skeletally immature joint

D. Howe, J. D. Thompson, S. D. Teeter, M. Easson, O. Barlow, E. H. Griffith, L. V. Schnabel, J. T. Spang, M. B. Fisher

This study examines how ACL injury severity affects degenerative changes in skeletally immature joints using a porcine model, finding that degeneration correlates with clinical Lachman grade.

bioRxiv · Nov 12, 2022 · PreprintBioengineering

The combination of mitogenic stimulation and cell cycle arrest induces senescence in equine and human cartilage explants

M. E. Copp, M. C. Flanders, R. Gagliardi, J. M. Gilbertie, G. A. Sessions, S. Chubinskaya, R. F. Loeser, L. V. Schnabel, B. O. Diekman

This study induces senescence in equine and human cartilage explants by combining mitogenic stimulation with cell cycle arrest, showing increased SA-β-gal activity.

bioRxiv · Mar 5, 2020Cell Biology