Author

Laurence Reutenauer

Lyon 1 Université

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:mouse· 2 Assay:proteomics· 2 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

Hélène M. PuccioiD· 2 shared Adele Hennick· 1 shared Alain MartelliiD· 1 shared Aurélie Eisenmann· 1 shared Débora Farina GonçalvesiD· 1 shared Emmanuelle SarziiD· 1 shared Laure Dall'Agnol· 1 shared Lenaic Marczak· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersCell Biology

Hélène M. PuccioORCID

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2 papers

Recent·Most cited

Partial Bypass of Frataxin Deficiency by ISCU M141I Restores Cytosolic and Nuclear FeS Cluster Assembly

Valentine Mosbach, Nunziata Maio, Lucie Hermet, Nadège Diedhiou, Adele Hennick, Laure Dall'Agnol, Laurence Reutenauer, Lenaic Marczak, Marie‐Christine Birling, Aurélie Eisenmann, Alain Martelli, Hélène M. Puccio

The paper shows that the ISCU M141I variant partially rescues Fe-S cluster assembly in FXN-deficient cells, preserving nuclear and cytosolic Fe-S proteins but not mitochondrial function.

Cell Death and Differentiation · Aug 6, 2026Peer-reviewedCell Biology

Characterization of a COQ8A-ataxia mouse model with E548K single-site mutation: distinct and comparable findings relative to a loss-of-function mutation

Débora Farina Gonçalves, Natacha Broyer, Maïté Carre-Pierrat, Emmanuelle Sarzi, Laurence Reutenauer, Hélène M. Puccio

This study characterizes a COQ8A-ataxia mouse model with a E548K mutation, finding no significant motor or cognitive impairments despite biochemical changes similar to a knockout model.

bioRxiv · Apr 23, 2025 · PreprintCell Biology