Interaction between BID and VDAC1 is required for mitochondrial demise and cell death in neurons
Sina Oppermann, Barbara Mertins, Lilja Meissner, Cornelius Krasel, Georgios Psakis, Philipp Reiß, Amalia M. Dolga, Nikolaus Plesnila, Moritz Bünemann, Lars‐Oliver Essen, Carsten Culmsee
The paper shows that BID interacts with VDAC1 to mediate mitochondrial permeabilization and neuronal cell death in response to stress.
bioRxiv · Sep 14, 2021 · PreprintMolecular Biology