Author

Sina Oppermann

Philipps University of MarburgORCID 0000-0002-9101-260XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Amalia M. DolgaiD· 1 shared Barbara Mertins· 1 shared Carsten CulmseeiD· 1 shared Cornelius KraseliD· 1 shared Georgios PsakisiD· 1 shared Lars‐Oliver EsseniD· 1 shared Lilja Meissner· 1 shared Moritz BünemanniD· 1 shared

Activity by Year

2021-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperMolecular Biology

Amalia M. DolgaORCID

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1 paper

Recent·Most cited

Interaction between BID and VDAC1 is required for mitochondrial demise and cell death in neurons

Sina Oppermann, Barbara Mertins, Lilja Meissner, Cornelius Krasel, Georgios Psakis, Philipp Reiß, Amalia M. Dolga, Nikolaus Plesnila, Moritz Bünemann, Lars‐Oliver Essen, Carsten Culmsee

The paper shows that BID interacts with VDAC1 to mediate mitochondrial permeabilization and neuronal cell death in response to stress.

bioRxiv · Sep 14, 2021 · PreprintMolecular Biology