Author

Carsten Culmsee

Philipps University of MarburgORCID 0000-0002-5121-5015Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Amalia M. DolgaiD· 1 shared Barbara Mertins· 1 shared Cornelius KraseliD· 1 shared Eva-Maria HanschmanniD· 1 shared Georgios PsakisiD· 1 shared Lars‐Oliver EsseniD· 1 shared Lena Hoffmann· 1 shared Lilja Meissner· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperMolecular Biology

Amalia M. DolgaORCID

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2 papers

Recent·Most cited

Cofilin1 oxidation links oxidative distress to mitochondrial demise and neuronal cell death

Lena Hoffmann, Marcel S. Waclawczyk, Stephan Tang, Eva-Maria Hanschmann, Manuela Gellert, Marco B. Rust, Carsten Culmsee

This study shows that cofilin1 oxidation contributes to mitochondrial dysfunction and neuronal death by linking oxidative stress to ferroptosis and excitotoxicity.

Cell Death and Disease · Oct 16, 2021Peer-reviewedNeuroscience

Interaction between BID and VDAC1 is required for mitochondrial demise and cell death in neurons

Sina Oppermann, Barbara Mertins, Lilja Meissner, Cornelius Krasel, Georgios Psakis, Philipp Reiß, Amalia M. Dolga, Nikolaus Plesnila, Moritz Bünemann, Lars‐Oliver Essen, Carsten Culmsee

The paper shows that BID interacts with VDAC1 to mediate mitochondrial permeabilization and neuronal cell death in response to stress.

bioRxiv · Sep 14, 2021 · PreprintMolecular Biology