Author

M. A. Brenden

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cancer Biology· 1

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Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Organism:human· 1 Organism:mouse· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Alarcon De La Lastra· 1 shared A. James· 1 shared B. L. Knolhoff· 1 shared C. Fu· 1 shared C. Zhou· 1 shared C. Zuo· 1 shared D. G. DeNardo· 1 shared G. D. Hogg· 1 shared

Activity by Year

2021-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

A. Alarcon De La Lastra

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1 paper

Recent·Most cited

Macrophage proliferation machinery leads to PDAC progression, but susceptibility to innate immunotherapy.

D. G. DeNardo, C. Zuo, J. M. Baer, B. L. Knolhoff, J. I. Belle, X. Liu, G. D. Hogg, C. Fu, N. L. Kingston, M. A. Brenden, A. Alarcon De La Lastra, P. B. Dodhiawala, C. Zhou, A. James, L. Ding, , J. D. Weber

This study shows that TAM proliferation in PDAC is driven by CSF1 from fibroblasts, leading to immunosuppression and tumor progression, but also increasing sensitivity to CD40 agonist therapy.

bioRxiv · Nov 8, 2021 · PreprintCancer Biology