Author

G. D. Hogg

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

B. L. Knolhoff· 2 shared c. zuo· 2 shared J. M. Baer· 2 shared X. Liu· 2 shared A. Alarcon De La Lastra· 1 shared A. James· 1 shared A. V. Galkin· 1 shared C. Fu· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

B. L. Knolhoff

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Context-dependent triggering of STING-interferon signaling by CD11b agonists supports anti-tumor immunity in mouse models and human cancer patients

X. Liu, G. D. Hogg, c. zuo, J. M. Baer, V. E. Lander, L. Kang, N. C. Borcherding, B. L. Knolhoff, R. E. Osterhout, A. V. Galkin, J. M. Bruey, L. L. Carter, C. Mpoy, J. K. Schwarz, H. Park, , D. DeNardo

This study identifies how CD11b agonists trigger STING-interferon signaling in tumor-associated macrophages to enhance anti-tumor immunity in mouse models and human cancer patients.

bioRxiv · Mar 22, 2022 · PreprintCancer Biology

Macrophage proliferation machinery leads to PDAC progression, but susceptibility to innate immunotherapy.

D. G. DeNardo, C. Zuo, J. M. Baer, B. L. Knolhoff, J. I. Belle, X. Liu, G. D. Hogg, C. Fu, N. L. Kingston, M. A. Brenden, A. Alarcon De La Lastra, P. B. Dodhiawala, C. Zhou, A. James, L. Ding, , J. D. Weber

This study shows that TAM proliferation in PDAC is driven by CSF1 from fibroblasts, leading to immunosuppression and tumor progression, but also increasing sensitivity to CD40 agonist therapy.

bioRxiv · Nov 8, 2021 · PreprintCancer Biology