Author

J. D. Weber

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 4 Approach:experimental assay· 3 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Approach:deep learning· 1

Frequent Co-Authors

K. A. Cottrell· 3 shared A. Alarcon De La Lastra· 1 shared A. James· 1 shared B. L. Knolhoff· 1 shared C. Fu· 1 shared C. Zhou· 1 shared C. Zuo· 1 shared C.-P. Kung· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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3 shared papersCancer Biology

K. A. Cottrell

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Macrophage proliferation machinery leads to PDAC progression, but susceptibility to innate immunotherapy.

D. G. DeNardo, C. Zuo, J. M. Baer, B. L. Knolhoff, J. I. Belle, X. Liu, G. D. Hogg, C. Fu, N. L. Kingston, M. A. Brenden, A. Alarcon De La Lastra, P. B. Dodhiawala, C. Zhou, A. James, L. Ding, , J. D. Weber

This study shows that TAM proliferation in PDAC is driven by CSF1 from fibroblasts, leading to immunosuppression and tumor progression, but also increasing sensitivity to CD40 agonist therapy.

bioRxiv · Nov 8, 2021 · PreprintCancer Biology

8-azaadenosine and 8-chloroadenosine are not selective inhibitors of ADAR

K. A. Cottrell, L. S. Torres, J. D. Weber

This paper shows that 8-azaadenosine and 8-chloroadenosine are not selective ADAR inhibitors, as they affect ADAR-dependent and independent cell lines equally and do not activate PKR or alter A-to-I editing.

bioRxiv · May 12, 2021Cancer Biology

ARF suppresses 5'-terminal oligopyrimidine mRNA translation

K. A. Cottrell, R. C. Chiou, J. D. Weber

The paper shows ARF suppresses 5'-terminal oligopyrimidine mRNA translation and identifies related regulators in cancer cells.

bioRxiv · Feb 7, 2020Cell Biology

ADAR1 editing dependency in triple-negative breast cancer

C.-P. Kung, K. A. Cottrell, S. Ryu, E. R. Bramel, R. D. Kladney, E. A. Bross, L. Maggi, J. D. Weber

This study shows ADAR1 is essential for TNBC cell proliferation and tumorigenesis, with its loss reducing proliferation and being rescued by IFNAR1 reduction.

bioRxiv · Jan 31, 2020Cancer Biology