Author

M. Bhalsen

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

A. Kimchi· 2 shared A. Savidor· 2 shared S. Bialik· 2 shared S. Meril· 2 shared C. Lior· 1 shared H. Belhanes Peled· 1 shared H. Birisi· 1 shared J. Krausz· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersCancer Biology

A. Kimchi

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Author profile →

2 papers

Recent·Most cited

Loss of EIF4G2 Mediates Aggressiveness in Distinct Human Endometrial Cancer Subpopulations with Poorer Survival Outcome in Patients

S. Meril, M. Muhlbauer Avni, C. Lior, M. Bhalsen, T. Olender, A. Savidor, J. Krausz, H. Belhanes Peled, H. Birisi, N. David, S. Bialik, R. Scherz-Shouval, Y. Ben David, A. Kimchi

This study shows that low EIF4G2 expression in Grade 2 endometrial cancer correlates with poor survival and drug resistance, suggesting it as a potential biomarker.

bioRxiv · Sep 14, 2023Cancer Biology

Loss-of-function cancer-associated mutations in the EIF4G2 non-canonical translation initiation factor

S. Meril, M. Bhalsen, M. Eisenstein, A. Savidor, Y. Levin, S. Bialik, S. Pietrokovski, A. Kimchi

The paper investigates EIF4G2 mutations in cancer, showing some mutations impair its non-canonical translation initiation function, suggesting potential tumor suppressor roles.

bioRxiv · Aug 22, 2023Cancer Biology