Author

S. Bialik

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 4

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 4 Organism:human· 4 Assay:proteomics· 4 Research Focus:cell type annotation· 3 Research Focus:differential expression· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

A. Kimchi· 4 shared A. Savidor· 3 shared M. Bhalsen· 2 shared N. P. Dekel-Bird· 2 shared N. Yaeli-Slonim· 2 shared S. Meril· 2 shared T. Olender· 2 shared V. Levin-Salomon· 2 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

4 shared papersCancer Biology

A. Kimchi

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Loss of EIF4G2 Mediates Aggressiveness in Distinct Human Endometrial Cancer Subpopulations with Poorer Survival Outcome in Patients

S. Meril, M. Muhlbauer Avni, C. Lior, M. Bhalsen, T. Olender, A. Savidor, J. Krausz, H. Belhanes Peled, H. Birisi, N. David, S. Bialik, R. Scherz-Shouval, Y. Ben David, A. Kimchi

This study shows that low EIF4G2 expression in Grade 2 endometrial cancer correlates with poor survival and drug resistance, suggesting it as a potential biomarker.

bioRxiv · Sep 14, 2023Cancer Biology

Loss-of-function cancer-associated mutations in the EIF4G2 non-canonical translation initiation factor

S. Meril, M. Bhalsen, M. Eisenstein, A. Savidor, Y. Levin, S. Bialik, S. Pietrokovski, A. Kimchi

The paper investigates EIF4G2 mutations in cancer, showing some mutations impair its non-canonical translation initiation function, suggesting potential tumor suppressor roles.

bioRxiv · Aug 22, 2023Cancer Biology

The Serine Protease HtrA2 mediates radiation-induced senescence in cancer cells

L. Hammer, V. Levin-Salomon, N. Yaeli-Slonim, M. Weiss, N. P. Dekel-Bird, T. Olender, Z. Porat, S. Winograd-Katz, A. Savidor, Y. Levin, S. Bialik, B. Geiger, A. Kimchi

This study identifies HtrA2 as a mediator of radiation-induced senescence in cancer cells, showing its role in proliferation arrest and vimentin filament reorganization.

bioRxiv · Feb 17, 2021Cancer Biology

A functional pre-screening platform for identifying points of vulnerability in the cell death map of human melanoma tumors

N. P. Dekel-Bird, S. Bialik, O. Itzhaki, T. M. Salame, N. Yaeli-Slonim, V. Levin-Salomon, S. K. Dasari, M. Besser, A. Kimchi

The paper develops a prescreening platform to identify tumor-specific vulnerabilities in melanoma cell death networks, improving targeted therapy by reducing drug resistance and relapse.

bioRxiv · Nov 11, 2020Cancer Biology