Author

M. D. Baruffi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1

Frequent Co-Authors

R. D. Cummings· 2 shared S. R. Stowell· 2 shared A. E. C. Seixas Azzolini· 1 shared C. R. d. B. Cardoso· 1 shared D. d. S. Lima-Junior· 1 shared D. G. Cerri· 1 shared H. Sales-Campos· 1 shared I. d. C. Kettelhut· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersCell Biology

R. D. Cummings

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Endogenous Galectin-3 is Required for Skeletal Muscle Repair

D. G. Cerri, L. C. Rodrigues, V. M. Alves, J. Machado, V. A. F. Bastos, I. d. C. Kettelhut, L. C. Alberici, S. R. Stowell, M. C. R. Costa, R. D. Cummings, M. D. Baruffi

This study shows that endogenous galectin-3 is essential for skeletal muscle repair and myogenic programming in mice.

bioRxiv · Oct 1, 2020Cell Biology

Galectin-1-deficient mice are protected against Trypanosoma cruzi infection through altered neutrophil migration and production of reactive oxygen species and nitric oxide

T. B. Riul, H. Sales-Campos, D. d. S. Lima-Junior, A. E. C. Seixas Azzolini, C. R. d. B. Cardoso, S. R. Stowell, R. D. Cummings, Y. M. L. Valim, M. D. Baruffi

This study shows that Galectin-1 deficiency in mice reduces T. cruzi parasitemia and increases survival by enhancing neutrophil migration and reactive oxygen species/nitric oxide production.

bioRxiv · Sep 11, 2020 · PreprintImmunology