Author

M. Köbel

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 1 Pathology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Albert· 1 shared A. Asante· 1 shared A. Hamade· 1 shared A. Lum· 1 shared B. C. Gilks· 1 shared B. J. Laight· 1 shared B. Tessier-Cloutier· 1 shared D. Chiu· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperPathology

A. Albert

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

PTEN and BRCA1 tumor suppressor loss associated tumor immune microenvironment exhibits differential response to therapeutic STING pathway activation in a murine model of ovarian cancer

N. Shakfa, D. Li, G. Conseil, E. D. Lightbody, J. Wilson-Sanchez, A. Hamade, S. Chenard, B. J. Laight, A. Asante, K. Tyryshkin, M. Köbel, M. Koti

This study examines how PTEN and BRCA1 loss in ovarian cancer affects tumor immune microenvironment response to STING pathway activation, showing improved survival in PTEN-deficient tumors with STING therapy.

bioRxiv · Jun 27, 2022 · PreprintImmunology

Oncogenic Mutations in Histologically Normal Endometrium: The New Normal?

V. Lac, T. M. Nazeran, B. Tessier-Cloutier, R. Aguirre-Hernandez, A. Albert, A. Lum, J. Khattra, T. Praetorius, M. Mason, D. Chiu, M. Köbel, P. J. Yong, B. C. Gilks, M. S. Anglesio, D. G. Huntsman

This study finds that over 50% of histologically normal endometrium samples contain somatic driver mutations, including KRAS, PIK3CA, FGFR2, and PTEN loss, with prevalence increasing with age.

bioRxiv · Feb 26, 2019Pathology