Author

D. Chiu

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cancer Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Organism:E. coli· 1 Approach:statistical model· 1

Frequent Co-Authors

D. G. Huntsman· 2 shared A. Albert· 1 shared A. C. Stankes· 1 shared A. Jeyasekharan· 1 shared A. Karnezis· 1 shared A. Lum· 1 shared A. S. Sedukhina· 1 shared B. C. Gilks· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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2 shared papersCancer Biology

D. G. Huntsman

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Rewiring native post-transcriptional carbon regulators to build multi-layered genetic circuits and optimize engineered microbes for bioproduction

T. R. Simmons, G. Partipilo, A. C. Stankes, R. Srivastava, R. Buchser, D. Chiu, B. K. Keitz, L. M. Contreras

This paper rewires the Csr Network in E. coli to create tunable genetic circuits for post-transcriptional control, achieving three-fold increased mevalonate production.

bioRxiv · Oct 4, 2023 · PreprintBioengineering

Primary platinum resistance and immune exclusion in ovarian carcinomas with high expression of the homologous recombination mediator RAD51

M. M. Hoppe, T. Z. Tan, S. Lie, S. Lim, J. P. Yeong, J. D. Wardyn, P. Jaynes, D. G. Lim, B. N. Pang, A. Karnezis, D. Chiu, S. Leung, D. G. Huntsman, A. S. Sedukhina, K. Sato, , A. Jeyasekharan

This study shows that high RAD51 expression in ovarian cancer is linked to poor survival and platinum resistance, particularly in HRD-negative tumors, and correlates with immune exclusion.

bioRxiv · Jun 9, 2020Cancer Biology

Oncogenic Mutations in Histologically Normal Endometrium: The New Normal?

V. Lac, T. M. Nazeran, B. Tessier-Cloutier, R. Aguirre-Hernandez, A. Albert, A. Lum, J. Khattra, T. Praetorius, M. Mason, D. Chiu, M. Köbel, P. J. Yong, B. C. Gilks, M. S. Anglesio, D. G. Huntsman

This study finds that over 50% of histologically normal endometrium samples contain somatic driver mutations, including KRAS, PIK3CA, FGFR2, and PTEN loss, with prevalence increasing with age.

bioRxiv · Feb 26, 2019Pathology