Author

M. Martinez-Gallo

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Assay:DNA methylation· 3 Approach:experimental assay· 3 Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:chromatin & epigenetic regulation· 2 Approach:statistical model· 2 Research Focus:cell-cell communication· 1

Frequent Co-Authors

E. Ballestar· 3 shared F. Catala-Moll· 2 shared J. Rodriguez-Ubreva· 2 shared O. Morante-Palacios· 2 shared A. Alvarez-Prado· 1 shared A. Durandy· 1 shared A. G. Ferrete-Bonastre· 1 shared A. Garcia-Gomez· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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3 shared papersImmunology

E. Ballestar

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Divergent Epigenetic and Transcriptomic Reprogramming of Monocyte Subpopulations in Systemic Lupus Erythematosus

A. G. Ferrete-Bonastre, M. Martinez-Gallo, O. Morante-Palacios, C. L. Calvillo, J. Calafell-Segura, J. Rodriguez-Ubreva, M. Esteller, J. Cortes-Hernandez, E. Ballestar

This study identifies epigenetic and transcriptomic changes in SLE monocyte subsets, linking them to disease activity and function.

bioRxiv · Dec 7, 2023 · PreprintImmunology

The JAK2-STAT pathway epigenetically regulates tolerized genes during the first encounter with bacterial antigens

C. Lorente-Sorolla, O. Morante-Palacios, A. Garcia-Gomez, L. Ciudad, F. Catala-Moll, A. Ruiz-Sanmartin, M. Martinez-Gallo, R. Ferrer-Roca, J. C. Ruiz-Rodriguez, D. Alvarez-Errico, E. Ballestar

The paper identifies JAK2-STAT pathway involvement in epigenetic regulation of tolerized genes during first bacterial antigen exposure, showing its role in modulating inflammatory responses.

bioRxiv · Oct 16, 2020 · PreprintImmunology

Activation-induced deaminase is critical for the establishment of DNA methylation patterns prior to the germinal center reaction

F. Catala-Moll, D. Weichenhan, P. Lutsik, A. Alvarez-Prado, J. Rodriguez-Ubreva, C. Klemann, C. Speckmann, H. Abolhassani, M. Martinez-Gallo, R. Dieli-Crimi, P. Soler-Palacin, S. Kracker, L. Hammarstrom, A. Durandy, B. Grimbacher, , E. Ballestar

This study investigates AID's role in DNA methylation defects in HIGM2 B cells, finding that AID is not directly involved in active demethylation but TET enzymes are, and that methylation defects are linked to premature BCR activation.

bioRxiv · Aug 16, 2019Immunology