Author

M. S. Remedi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:metabolism & bioenergetics· 1 Organism:mouse· 1 Organism:zebrafish· 1

Frequent Co-Authors

A. Ustione· 1 shared C. G. Nichols· 1 shared E. Egan· 1 shared J. M. Ikle· 1 shared N. W. York· 1 shared R. C. Tryon· 1 shared S. S. Singareddy· 1 shared Z. A. Shyr· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

9 top collaborators · 16 mutual co-author links

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1 shared paperCell Biology

A. Ustione

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2 papers

Recent·Most cited

Genome-edited zebrafish model of ABCC8 loss-of-function disease

J. M. Ikle, R. C. Tryon, S. S. Singareddy, N. W. York, M. S. Remedi, C. G. Nichols

This paper creates a zebrafish model of ABCC8 loss-of-function disease by introducing a SUR1 truncation mutation and shows it mimics key features of congenital hyperinsulinism seen in mice.

bioRxiv · Oct 10, 2021Cell Biology

SGLT2 inhibitors therapy protects glucotoxicity-induced β-cell failure in a mouse model of human KATP-induced diabetes trough mitigation of oxidative and ER stress

Z. A. Shyr, Z. Yan, A. Ustione, E. Egan, M. S. Remedi

This study shows SGLT2 inhibitors reduce oxidative and ER stress, restoring β-cell function in early-stage NDM mice but not when β-cell mass and identity are already lost.

bioRxiv · Sep 17, 2021 · PreprintCell Biology