Author

M. Teruel

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Assay:scRNA-seq· 2 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Z. Bahrami-Nejad· 3 shared M. L. Zhao· 2 shared Z.-b. Zhang· 2 shared A. Dunn· 1 shared A. Rabiee· 1 shared A. Shah· 1 shared B. Taylor· 1 shared C. Miller· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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3 shared papersCell Biology

Z. Bahrami-Nejad

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3 papers

Recent·Most cited

The circadian clock mediates daily bursts of cell differentiation by periodically restricting cell differentiation commitment

Z.-b. Zhang, J. Sinha, Z. Bahrami-Nejad, M. Teruel

The paper shows that the circadian clock drives daily bursts of adipocyte differentiation by restricting differentiation commitment to specific rest periods through clock-regulated gene expression.

bioRxiv · May 17, 2022Cell Biology

Early enforcement of cell identity by a functional component of the terminally differentiated state

Z. Bahrami-Nejad, T. Chen, S. Tholen, Z.-b. Zhang, A. Rabiee, M. L. Zhao, M. Teruel

The paper shows that FABP4, a core component of mature adipocytes, drives differentiation by forming positive feedback loops with PPARG, enabling irreversible commitment to the adipocyte state.

bioRxiv · Jan 3, 2020 · PreprintCell Biology

Loss of adipocyte identity through synergistic repression of PPARγ by TGF-β and mechanical stress

E. Bielczyk-Maczynska, B. Taylor, C. Miller, M. L. Zhao, A. Shah, Z. Bahrami-Nejad, A. Dunn, M. Teruel

The paper shows that TGF-β and mechanical stress together repress PPARγ, causing adipocytes to lose identity and convert into myofibroblasts in a fibrosis model.

bioRxiv · Apr 11, 2019Cell Biology