Author

M. Willenbrock

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Akalin· 1 shared A. B. Tufan· 1 shared B. Uyar· 1 shared C. A. Schmitt· 1 shared C. Scheidereit· 1 shared I. Krahn· 1 shared K. Schleich· 1 shared L. von Hoff· 1 shared

Activity by Year

2019-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCell Biology

A. Akalin

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1 paper

Recent·Most cited

A novel IKK- and proteasome-independent mechanism of RelA activation triggers senescence associated secretome via transcriptional repression of NFKBIA

M. Kolesnichenko, N. Mikuda, U. E. Höpken, M. Milanovic, A. B. Tufan, B. Uyar, W. Sun, K. Schleich, L. von Hoff, M. Willenbrock, I. Krahn, S. Jungmann, M. Hinz, A. Akalin, S. Lee, , C. Scheidereit

The paper identifies a novel IKK- and proteasome-independent mechanism of RelA activation in senescent cells that drives the senescence-associated secretome through transcriptional repression of NFKBIA.

bioRxiv · Dec 19, 2019Cell Biology