Author

Madison M. Reeves

Mayo Clinic

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Dennis W. DicksoniD· 2 shared Mercedes PrudencioiD· 2 shared Adam N. TrautwigiD· 1 shared Aleksandar Rajkovic· 1 shared Alyssa C. WalkeriD· 1 shared Anand Narajan· 1 shared Andrew StrangeiD· 1 shared Aron S. BuchmaniD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersNeuroscience

Dennis W. DicksonORCID

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2 papers

Recent·Most cited

TDP-43 subtypes shape transcriptomic signatures in Alzheimer's disease

Xiaojie Wang, Alyssa C. Walker, Madison M. Reeves, Jesús González Bejarano, Yuping Song, Judy H. Dunmore, Mei Yue, Bailey D. Rawlinson, Erica Engelberg-Cook, Michael A. Deture, Raul Baena Tome, Anand Narajan, Jordan L. Bartfield, Neill R. Graff‐Radford, Bradley F. Boeve, , Mercedes Prudencio

The paper decouples TDP-43 and Tau pathology in Alzheimer's disease by analyzing transcriptomic profiles and identifies subtype-specific transcriptional signatures.

bioRxiv · Jun 26, 2026 · PreprintNeuroscience

TDP-43 toxic gain of function links ALS, FTD and Alzheimers Disease through splicing dysregulation

Welmoed van Zuiden, Thea Deborah Meimoun, Bar Chen, Aviad Siany, Bar Abarbanel, Lihi Moshe, Nancy‐Sarah Yacovzada, Emmanuel Amzallag, Aleksandar Rajkovic, Liat Alyagor, Eviatar Weizman, Manuela Neumann, Colin Smith, Karina McDade, Andrew Strange, , Eran Hornstein

This study identifies TDP-43-driven splicing dysregulation as a common mechanism in ALS, FTD, and Alzheimer's disease, linking them through mis-spliced mRNAs like APP.

bioRxiv · Apr 20, 2025 · PreprintNeuroscience