Author

Megumi Hayashi

Kumamoto University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Keiko Ueno-ShutoiD· 2 shared Keisuke Kawano· 2 shared Mai Uemura· 2 shared Mary Ann SuicoiD· 2 shared Noriki TAKAHASHIiD· 2 shared Ryunosuke Nakashima· 2 shared Tomoki Kishimoto· 2 shared Tsuyoshi ShutoiD· 2 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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Curved chords = mutual co-authorship

2 shared papersImmunology

Keiko Ueno-ShutoORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Gingipain-containing products from Porphyromonas gingivalis promote epithelial CCL20 signaling and γδ T-cell accumulation in COPD-like airways

Keisuke Kawano, Noriki TAKAHASHI, Tomoki Kishimoto, Toru Kariu, Yukio Fujiwara, Mai Uemura, Kota Nakajima, Nodoka Kinjo, Keiko Ueno-Shuto, Ryunosuke Nakashima, Megumi Hayashi, Mary Ann Suico, Tsuyoshi Shuto

This paper shows that Porphyromonas gingivalis gingipain products induce CCL20 signaling and γδ T-cell accumulation in COPD-like airways via PAR-2 activation.

Journal of Pharmacological Sciences · Oct 26, 2026Peer-reviewedImmunology

Dietary lipid stress aggravates obstructive lung injury involving epithelial IGF-1-Akt dysfunction and epithelial-endothelial crosstalk

Choyo Ogasawara, Hirofumi Nohara, Tomoki Kishimoto, Keisuke Kawano, Kenji Watanabe, Kentaro Oniki, Yukio Fujiwara, Ryunosuke Nakashima, Shunsuke Kamei, Noriki TAKAHASHI, Megumi Hayashi, Ayami Fukuyama, Mai Uemura, Keiko Ueno-Shuto, Junji Saruwatari, , Tsuyoshi Shuto

This study shows that high-fat diet worsens obstructive lung injury by impairing epithelial IGF-1-Akt signaling and promoting epithelial-endothelial interactions.

bioRxiv · Sep 3, 2026 · PreprintMolecular Biology