Author

Michael Moedritzer

University of Kansas Medical Center

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Developmental Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:development & morphogenesis· 1 Assay:imaging· 1

Frequent Co-Authors

Irfan SaadiiD· 3 shared Jeremy P. Goering· 3 shared Marta StetsiviD· 3 shared András CzirókiD· 2 shared Brittany M. Hufft‐MartineziD· 2 shared Dona Greta Isai· 2 shared Madison K. Rickabaugh· 2 shared Zaid Umar· 2 shared

Activity by Year

2021-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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3 shared papersDevelopmental Biology

Irfan SaadiORCID

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3 papers

Recent·Most cited

Genetic interaction of Specc1l and Thm1 reveals cytoskeletal - ciliary crosstalk

Brittany M. Hufft‐Martinez, Dana N Thalman, An J. Tran, Jeremy P. Goering, Marta Stetsiv, Michael Moedritzer, Sarah C Wilson, Henry H. Wang, Pamela Vivian Tran, Irfan Saadi

The paper shows that SPECC1L regulates F-actin levels, which in turn affects cilia length and palate development, with a genetic interaction with Thm1.

bioRxiv · Nov 3, 2025 · PreprintCell Biology

Novel insights into the fundamentals of palatal shelf elevation dynamics in normal mouse embryos

Jeremy P. Goering, Michael Moedritzer, Marta Stetsiv, Dona Greta Isai, Brittany M. Hufft‐Martinez, Zaid Umar, Madison K. Rickabaugh, Paul Keselman, Munish Chauhan, William Miles Brooks, Kenneth J. Fischer, András Czirók, Irfan Saadi

This study reveals the dynamic timing and sex-specific differences in palatal shelf elevation in mouse embryos using high-resolution imaging.

bioRxiv · Jun 2, 2022 · PreprintDevelopmental Biology

In-frame deletion of SPECC1L microtubule binding domain results in embryonic tissue movement and fusion defects

Jeremy P. Goering, Luke W. Wenger, Marta Stetsiv, Michael Moedritzer, Everett G. Hall, Dona Greta Isai, Brittany Jack, Zaid Umar, Madison K. Rickabaugh, András Czirók, Irfan Saadi

This study shows that in-frame deletions in the SPECC1L CCD2 domain cause embryonic tissue movement and fusion defects, leading to cleft palate and exencephaly, and identifies CCD2 as a gain-of-function mutation.

bioRxiv · Jan 28, 2021 · PreprintDevelopmental Biology