Author

Henry H. Wang

University of Kansas Medical Center

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genetics· 3 Cell Biology· 1 Developmental Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 5 Organism:mouse· 5 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Pamela Vivian TraniD· 5 shared Tana S. PottorfiD· 4 shared Wei WangiD· 4 shared Bailey A. AllardiD· 3 shared Brittany M. Hufft‐MartineziD· 2 shared Damon T. Jacobs· 2 shared Joseph T. Cornelius· 2 shared Luciane M. Silva· 2 shared

Activity by Year

2020-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

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5 shared papersGenetics

Pamela Vivian TranORCID

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Author profile →

5 papers

Recent·Most cited

Genetic interaction of Specc1l and Thm1 reveals cytoskeletal - ciliary crosstalk

Brittany M. Hufft‐Martinez, Dana N Thalman, An J. Tran, Jeremy P. Goering, Marta Stetsiv, Michael Moedritzer, Sarah C Wilson, Henry H. Wang, Pamela Vivian Tran, Irfan Saadi

The paper shows that SPECC1L regulates F-actin levels, which in turn affects cilia length and palate development, with a genetic interaction with Thm1.

bioRxiv · Nov 3, 2025 · PreprintCell Biology

Intraflagellar transport-A deficiency attenuates ADPKD in a renal tubular- and maturation-dependent manner

Wei Wang, Luciane M. Silva, Henry H. Wang, Matthew A. Kavanaugh, Tana S. Pottorf, Bailey A. Allard, Damon T. Jacobs, Ruochen Dong, Joseph T. Cornelius, Aakriti Chaturvedi, Katherine I. Swenson-Fields, Timothy A. Fields, Michele T. Pritchard, Madhulika Sharma, Chad M Slawson, , Pamela Vivian Tran

This study shows that deleting the IFT-A gene Thm1 in ADPKD mouse models has development-specific effects on kidney cysts, inflammation, and cilia length, suggesting environmental factors influence ciliary homeostasis and disease progression.

Kidney International · May 26, 2022Peer-reviewedGenetics

IFT-A deficiency in juvenile mice impairs biliary development and exacerbates ADPKD liver disease

Wei Wang, Tana S. Pottorf, Henry H. Wang, Ruochen Dong, Matthew A. Kavanaugh, Joseph T. Cornelius, Katie L. Dennis, Udayan M. Apte, Michele T. Pritchard, Madhulika Sharma, Pamela Vivian Tran

This study shows that Thm1 deletion in juvenile Pkd2 mice worsens biliary development and PLD, while adult Thm1 deletion has no effect, highlighting distinct roles of IFT-A and IFT-B in liver disease.

The Journal of Pathology · Apr 26, 2021Peer-reviewedGenetics

Thm2 interacts with paralog, Thm1, and sensitizes to Hedgehog signaling in postnatal skeletogenesis

Bailey A. Allard, Wei Wang, Tana S. Pottorf, Hammad Mumtaz, Brittany M. Hufft‐Martinez, Henry H. Wang, Luciane M. Silva, Damon T. Jacobs, Jinxi Wang, Erin Ealba Bumann, Pamela Vivian Tran

This study identifies Thm2 as a novel locus that interacts with Thm1 and sensitizes to Hedgehog signaling in postnatal skeletal development, leading to osteochondrodysplasia.

Cellular and Molecular Life Sciences · Mar 8, 2021Peer-reviewedDevelopmental Biology

Genetic interaction of mammalian IFT-A paralogs regulates cilia disassembly, ciliary protein trafficking, Hedgehog signaling and embryogenesis

Wei Wang, Bailey A. Allard, Tana S. Pottorf, Henry H. Wang, Jay L. Vivian, Pamela Vivian Tran

This study investigates the roles of Thm1 and Thm2 in cilia function and embryogenesis using mouse embryonic fibroblasts, revealing their distinct and overlapping contributions to ciliary dynamics and signaling.

The FASEB Journal · Mar 13, 2020Peer-reviewedGenetics