Author

Michael W. Shultis

Albert Einstein College of Medicine

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Microbiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Christopher R. HineriD· 1 shared Claire V. MulhollandiD· 1 shared Jinhua Cui· 1 shared M Chen· 1 shared Michael BerneyiD· 1 shared Samantha Johnson· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

6 top collaborators · 15 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperMicrobiology

Christopher R. HinerORCID

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

A new Persister Enrichment Model in Mycobacterium tuberculosis links multidrug persistence to drug sequencing and maltokinase-dependent carbon distribution.

Michael W. Shultis, Claire V. Mulholland, Jinhua Cui, M Chen, Samantha Johnson, Christopher R. Hiner, Michael Berney

This paper introduces a Persister Enrichment Model (PEM) to study multidrug persistence in M. tuberculosis, showing that drug sequencing and maltokinase-dependent carbon distribution influence antibiotic tolerance.

bioRxiv · May 29, 2026 · PreprintMicrobiology