Author

N. Posnack

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Assay:electrophysiology· 1 Organism:mouse· 1

Frequent Co-Authors

D. Guerrelli· 2 shared B. L. Cooper· 1 shared C. Mulvany· 1 shared J. Pressman· 1 shared J. Romanowicz· 1 shared L. Swift· 1 shared M. Ramadan· 1 shared M. Reilly· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersPhysiology

D. Guerrelli

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

hiPSC-CM Electrophysiology: Impact of Temporal Changes and Study Parameters on Experimental Reproducibility

D. Guerrelli, J. Pressman, N. Posnack

This study examines how temporal changes and experimental parameters affect the electrophysiology of hiPSC-CMs, highlighting variability in beating rate, field potential duration, and drug responsiveness.

bioRxiv · Oct 2, 2023Physiology

Comparative cardiotoxicity assessment of bisphenol chemicals and estradiol using human induced pluripotent stem cell-derived cardiomyocytes

B. L. Cooper, S. Salameh, N. Posnack

This study compares the cardiotoxic effects of bisphenol chemicals and estradiol on human induced pluripotent stem cell-derived cardiomyocytes, finding E2 to be the most potent and BPS the least potent disruptor.

bioRxiv · Sep 13, 2023Pharmacology & Toxicology

Chronic Perinatal Hypoxia Delays Cardiac Maturation in a Mouse Model for Cyanotic Congenital Heart Disease

J. Romanowicz, Z. Dhari, D. Guerrelli, C. Mulvany, M. Reilly, L. Swift, N. Vasandani, M. Ramadan, N. Ishibashi, N. Posnack

This study examines the effects of chronic perinatal hypoxia on cardiac development in a mouse model of cyanotic congenital heart disease, finding persistent electrophysiological and functional impairments.

bioRxiv · May 28, 2020Physiology