Author

N. Ruparelia

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Clerk· 1 shared A. Whyte· 1 shared C. McKenna· 1 shared D. N. Meijles· 1 shared H. O. Alharbi· 1 shared J. J. Cull· 1 shared K. Allen-Redpath· 1 shared L. Dymott· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperPhysiology

A. Clerk

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2 papers

Recent·Most cited

Striatin plays a major role in angiotensin II-induced cardiomyocyte and cardiac hypertrophy in mice in vivo.

J. J. Cull, S. T. Cooper, H. O. Alharbi, S. Chothani, O. J. Rackham, D. N. Meijles, P. R. Dash, R. Kerkela, N. Ruparelia, P. H. Sugden, A. Clerk

This paper shows that striatin (STRN) in cardiomyocytes is necessary for angiotensin II-induced cardiac hypertrophy and remodelling in mice, but not STRN3.

bioRxiv · Oct 21, 2023 · PreprintPhysiology

Extracellular vesicles from patients with coronary artery disease (CAD) demonstrate enhanced pro-coagulatory activity.

K. Allen-Redpath, S. Soyama, R. Zhou, A. Whyte, S. Mark, L. Dymott, M. Brunton, R. Fischer, S. Hester, C. McKenna, N. Ruparelia, P. Yaqoob

This study finds that extracellular vesicles from CAD patients show increased pro-coagulatory activity despite similar numbers to controls, suggesting a role in CAD pathogenesis.

bioRxiv · Sep 27, 2023 · PreprintPhysiology