Author

Nadezhda Igolkina

University of Vienna

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Evolutionary Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:development & morphogenesis· 1 Organism:Drosophila· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Almorò ScarpaiD· 1 shared Anna HaideriD· 1 shared Gonzalo SabarísiD· 1 shared Josefa GonzáleziD· 1 shared Matthew BeaumontiD· 1 shared Robert KofleriD· 1 shared Rossella Damiano· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

7 top collaborators · 21 mutual co-author links

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1 shared paperEvolutionary Biology

Almorò ScarpaORCID

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1 paper

Recent·Most cited

A novel hybrid dysgenesis system in Drosophila melanogaster suggests that transposons may contribute to speciation

Almorò Scarpa, Anna Haider, Matthew Beaumont, Nadezhda Igolkina, Rossella Damiano, Gonzalo Sabarís, Josefa González, Robert Kofler

This study identifies a novel hybrid dysgenesis system in Drosophila melanogaster caused by Transib1, showing strong and temperature-tolerant effects, and demonstrates bidirectional gene flow barriers using two TEs.

bioRxiv · Oct 30, 2026 · PreprintEvolutionary Biology