Author

Nanci S. Kane

Rutgers, The State University of New Jersey

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Organism:C. elegans· 2 Assay:bulk RNA-seq· 1 Assay:ChIP-seq· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:metabolomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Mehul VoraiD· 2 shared Aparna Prashar· 1 shared Christopher RongoiD· 1 shared Jing LiniD· 1 shared Premal ShahiD· 1 shared Richard W. Padgett· 1 shared Ryan J. Gleason· 1 shared Stephanie M. PyonteckiD· 1 shared

Activity by Year

2018-2021 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 21 mutual co-author links

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2 shared papersGenetics

Mehul VoraORCID

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2 papers

Recent·Most cited

The Hypoxia Response Pathway Promotes PEP Carboxykinase Expression And Gluconeogenesis

Mehul Vora, Stephanie M. Pyonteck, Tarmie L. Matlack, Aparna Prashar, Nanci S. Kane, Premal Shah, Christopher Rongo

The paper shows HIF-1 promotes gluconeogenesis by upregulating PCK-1 in C. elegans, revealing a new role for HIF-1 beyond glycolysis.

bioRxiv · May 4, 2021 · PreprintGenetics

C. elegans models of Marfan and Marfan-like Syndromes reveal trafficking defects of the type II TGFβ receptor as a potential novel disease mechanism

Jing Lin, Mehul Vora, Nanci S. Kane, Ryan J. Gleason, Richard W. Padgett

This paper investigates Marfan and Marfan-like syndromes in C. elegans, revealing that type II TGFβ receptor mutations cause mis-trafficking rather than loss of function, suggesting a novel disease mechanism.

bioRxiv · Dec 27, 2018 · PreprintCancer Biology