Author

Naoko Ishibashi

Tokyo Metropolitan Institute of Medical Science

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cell Biology· 1

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Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Assay:proteomics· 1

Frequent Co-Authors

Akinori EndoiD· 1 shared Chikage Takahashi· 1 shared Keiji TanakaiD· 1 shared Koji YamanoiD· 1 shared Yasumasa NishitoiD· 1 shared Yukiko YoshidaiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

6 top collaborators · 15 mutual co-author links

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1 shared paperCell Biology

Akinori EndoORCID

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1 paper

Recent·Most cited

Ubiquitination-activated TAB-TAK1-IKK-NF-kB axis modulates gene expression for cell survival in the lysosomal damage response

Akinori Endo, Chikage Takahashi, Naoko Ishibashi, Yasumasa Nishito, Koji Yamano, Keiji Tanaka, Yukiko Yoshida

The paper identifies the TAB-TAK1-IKK-NF-κB axis as a ubiquitin-regulated pathway activated by lysosomal damage, promoting cell survival through anti-apoptotic and intercellular signaling genes.

bioRxiv · Mar 26, 2025Cell Biology