Author

O. GARNIER

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:gene regulatory network inference· 2 Organism:mouse· 2 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

D. Martin· 3 shared I. VILGRAIN· 3 shared A. DURAND· 2 shared A. FERTIN· 2 shared C. Battail· 2 shared F. JEANNERET· 2 shared G. CARPENTIER· 2 shared S. BERNDT· 2 shared

Activity by Year

2023-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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3 shared papersCell Biology

D. Martin

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Efficient fine-tuning of endothelial gene expression by Y to Phe mutation in the VE-cadherin gene

O. GARNIER, F. JEANNERET, A. DURAND, A. FERTIN, C. Battail, S. BERNDT, G. CARPENTIER, D. Martin, I. VILGRAIN

The paper investigates the effect of a Y685F mutation in VE-cadherin on endothelial gene expression, identifying 884 differentially expressed genes and altered signaling pathways in vivo.

bioRxiv · Apr 17, 2024 · PreprintCell Biology

Single tyrosine mutation in VE-cadherin modulates gene lung expressions: evidence for FOXF1 mediated S1PR1 upregulation to stabilize vessels in mice

O. GARNIER, F. JEANNERET, A. DURAND, A. FERTIN, D. MARTIN, S. BERNDT, G. CARPENTIER, C. BATTAIL, I. VILGRAIN

This study shows that a single tyrosine mutation in VE-cadherin increases S1PR1 expression via FOXF1 binding, stabilizing lung vessels in mice.

bioRxiv · Jul 28, 2023 · PreprintCell Biology

The enigma of the SARS-CoV-2 microcirculation dysfunction: evidence for modified endothelial junctions.

L. Bouillet, M. Benmarce, C. Guerin, L. Bouvet, O. Garnier, D. Martin, I. Vilgrain

This paper investigates how SARS-CoV-2 affects endothelial junctions by targeting VE-cadherin and ACE2, finding increased soluble fragments in severe COVID-19 patients.

bioRxiv · Apr 24, 2023 · PreprintBiochemistry