Author

C. Battail

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 2 Biochemistry· 1 Evolutionary Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:gene regulatory network inference· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

F. JEANNERET· 3 shared A. DURAND· 2 shared A. FERTIN· 2 shared D. Martin· 2 shared G. CARPENTIER· 2 shared I. VILGRAIN· 2 shared O. GARNIER· 2 shared S. BERNDT· 2 shared

Activity by Year

2017-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersCell Biology

F. JEANNERET

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Efficient fine-tuning of endothelial gene expression by Y to Phe mutation in the VE-cadherin gene

O. GARNIER, F. JEANNERET, A. DURAND, A. FERTIN, C. Battail, S. BERNDT, G. CARPENTIER, D. Martin, I. VILGRAIN

The paper investigates the effect of a Y685F mutation in VE-cadherin on endothelial gene expression, identifying 884 differentially expressed genes and altered signaling pathways in vivo.

bioRxiv · Apr 17, 2024 · PreprintCell Biology

Single tyrosine mutation in VE-cadherin modulates gene lung expressions: evidence for FOXF1 mediated S1PR1 upregulation to stabilize vessels in mice

O. GARNIER, F. JEANNERET, A. DURAND, A. FERTIN, D. MARTIN, S. BERNDT, G. CARPENTIER, C. BATTAIL, I. VILGRAIN

This study shows that a single tyrosine mutation in VE-cadherin increases S1PR1 expression via FOXF1 binding, stabilizing lung vessels in mice.

bioRxiv · Jul 28, 2023 · PreprintCell Biology

AB668, a novel highly selective protein kinase CK2 inhibitor with a distinct anti-tumor mechanism as compared to CX-4945 and SGC-CK2-1

A. Bancet, R. Frem, F. Jeanneret, A. Mularoni, P. Bazelle, C. Roelants, J.-G. Delcros, J.-F. Guichou, C. Pillet, I. Coste, T. Renno, C. Battail, C. Cochet, T. Lomberget, O. Filhol, I. Krimm

The paper introduces AB668, a selective CK2 inhibitor with a distinct anti-tumor mechanism compared to CX-4945 and SGC-CK2-1, showing apoptotic effects and pathway stimulation in cancer cells.

bioRxiv · Dec 16, 2022 · PreprintBiochemistry

Disentangling The Causes For Faster-X Evolution In Aphids

J. Jaquiery, J. Peccoud, T. Ouisse, F. Legeai, N. Prunier-Leterme, A. Gouin, P. Nouhaud, J. Brisson, R. Bickel, S. Purandare, J. Poulain, C. Battail, C. Lemaitre, L. Mieuzet, G. Le Trionnaire, , C. Rispe

This study investigates faster-X evolution in aphids by analyzing gene expression, substitution rates, and polymorphism data to determine the role of selection and drift.

bioRxiv · May 10, 2017Evolutionary Biology