Author

P. Ajitkumar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 5

Signature Assays, Organisms & Methods

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Approach:experimental assay· 5 Organism:human· 3 Organism:E. coli· 2 Research Focus:host-pathogen & immune response· 2 Organism:bacteria (other)· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

A. Pradhan· 3 shared K. Jakkala· 3 shared R. R. Nair· 3 shared D. Sharan· 2 shared S. Swaminath· 2 shared A. Paul· 1 shared A. S. N. Seshasayee· 1 shared A. Singh· 1 shared

Activity by Year

2017-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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3 shared papersMicrobiology

A. Pradhan

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Author profile →

5 papers

Recent·Most cited

Complete identity of the cis-asRNA StfZ and its influence on FtsZ protein level and cell division in Escherichia coli

D. Anand, K. Jakkala, R. R. Nair, D. Sharan, A. Pradhan, N. Mukkayyan, P. Ajitkumar

The paper identifies the complete sequence and promoters of the cis-antisense RNA StfZ in E. coli and shows it regulates FtsZ protein levels and cell division.

bioRxiv · Mar 8, 2022 · PreprintMicrobiology

The rifampicin-inactivating mono-ADP-ribosyl transferase of Mycobacterium smegmatis significantly influences reactive oxygen species levels in the actively growing cells

S. Swaminath, A. Pradhan, R. R. Nair, P. Ajitkumar

This study shows that the M. smegmatis Arr enzyme reduces reactive oxygen species levels in actively growing cells, and its absence increases ROS, leading to higher rifampicin resistance.

bioRxiv · Jan 10, 2020 · PreprintMicrobiology

Unveiling the physiological mechanisms that drive the emergence of antibiotic resisters from antibiotic persister population of mycobacteria

K. Jakkala, D. Sharan, R. R. Nair, A. Paul, A. Pradhan, P. Ajitkumar

The paper identifies sequential physiological events in mycobacteria that lead to antibiotic resistance emergence from persister populations, showing similar responses in other bacterial genera.

bioRxiv · Nov 18, 2019 · PreprintMicrobiology

Reduced Permeability to Rifampicin by Capsular Thickening as a Mechanism of Antibiotic Persistence in Mycobacterium tuberculosis

J. Sebastian, S. Swaminath, P. Ajitkumar

This study identifies reduced permeability to rifampicin via a thick outer layer (TOL) capsule as a mechanism for antibiotic persistence in Mycobacterium tuberculosis.

bioRxiv · May 1, 2019 · PreprintMicrobiology

Redox-Dependent Condensation Of the Mycobacterial Nucleoid By WhiB4

M. Chawla, M. Mehta, P. Parikh, S. Mishra, P. Shukla, M. Vij, K. Jakkala, P. Singh, P. AjitKumar, H. N. Verma, M. Ganguli, A. S. N. Seshasayee, A. Singh

This paper identifies WhiB4 as a redox-dependent nucleoid condensing protein in Mtb that links oxidative stress response with genome organization and gene expression.

bioRxiv · Nov 26, 2017Microbiology