Author

P. Brossart

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

D. Wolf· 2 shared A. Heine· 1 shared C. C. Kolbe· 1 shared C. Flores· 1 shared C. Krollmann· 1 shared C. Kurts· 1 shared E. Latz· 1 shared E. Tregel· 1 shared

Activity by Year

2024-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCancer Biology

D. Wolf

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2 papers

Recent·Most cited

NLRP3-induced systemic inflammation controls the development of JAK2V617F mutant myeloproliferative neoplasms

R.-M. Koerber, C. Krollmann, K. Cieslak, E. Tregel, T. H. Brümmendorf, S. Koschmieder, M. Griesshammer, I. Gütgemann, P. Brossart, R. Skoda, C. C. Kolbe, E. Latz, D. Wolf, L. L. Teichmann

This study shows that NLRP3 inflammasome activation drives JAK2V617F mutant myeloproliferative neoplasms by promoting inflammation, platelet production, and fibrosis, and that its inhibition can reverse disease features.

bioRxiv · Mar 9, 2024Cancer Biology

The CCR4/CCL17 axis drives intestinal acute Graft versus Host disease after allogeneic bone marrow transplantation

S. Schlaweck, M. Klesse, O. Schanz, S. K. Jorch, G. Kristiansen, M. Toma, C. Flores, J. Becker-Gotot, C. Kurts, P. Brossart, D. Wolf, A. Heine

This study shows that the CCR4/CCL17 axis promotes intestinal acute GvHD by recruiting T cells and increasing Gata3 expression, and identifies ruxolitinib as a potential therapeutic.

bioRxiv · Mar 2, 2024 · PreprintImmunology