Author

P. G. Hendrickson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Assay:proteomics· 1

Frequent Co-Authors

D. G. Kirsch· 2 shared A. R. Daniels· 1 shared C. M. Linardic· 1 shared D. Corcoran· 1 shared D. M. Cardona· 1 shared J. Blackmer· 1 shared J. Huang· 1 shared K. M. Oristian· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCancer Biology

D. G. Kirsch

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2 papers

Recent·Most cited

Expression of the CIC-DUX4 fusion oncoprotein mimics human CIC-rearranged sarcoma in genetically engineered mouse models

P. G. Hendrickson, K. M. Oristian, M. R. Browne, L. Lou, Y. Ma, D. M. Cardona, C. M. Linardic, D. G. Kirsch

This study generates genetically engineered mouse models of CIC-DUX4 sarcoma that spontaneously develop tumors and express the fusion protein, validating CIC-DUX4 as a transcriptional activator.

bioRxiv · Sep 26, 2023 · PreprintCancer Biology

p53 and RB Cooperate to Suppress Transposable Elements

O. M. Lopez, M. Zhang, P. G. Hendrickson, J. Huang, A. R. Daniels, J. Blackmer, L. Luo, L. D. Attardi, D. Corcoran, D. G. Kirsch

This study shows that loss of p53 and RB in mouse cells leads to epigenetic changes and upregulation of transposable elements, suggesting they work together to suppress transposons and prevent tumorigenesis.

bioRxiv · Feb 6, 2023 · PreprintCancer Biology