Author

L. D. Attardi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Assay:DNA methylation· 1 Organism:human· 1

Frequent Co-Authors

D. G. Kirsch· 2 shared L. Luo· 2 shared A. Ayyaz· 1 shared A. J. Minn· 1 shared A. M. Boutelle· 1 shared A. M. Kaiser· 1 shared A. R. Daniel· 1 shared A. R. Daniels· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersCell Biology

D. G. Kirsch

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

p53 promotes revival stem cells in the regenerating intestine after severe radiation injury

C. Morral, A. Ayyaz, H.-C. Kuo, M. Fink, I. Verginadis, A. R. Daniel, D. N. Burner, L. M. Driver, S. Satow, S. Hasapis, R. Ghinnagow, L. Luo, Y. Ma, L. D. Attardi, C. Koumenis, , D. G. Kirsch

bioRxiv · Apr 27, 2023Cell Biology

p53 and RB Cooperate to Suppress Transposable Elements

O. M. Lopez, M. Zhang, P. G. Hendrickson, J. Huang, A. R. Daniels, J. Blackmer, L. Luo, L. D. Attardi, D. Corcoran, D. G. Kirsch

This study shows that loss of p53 and RB in mouse cells leads to epigenetic changes and upregulation of transposable elements, suggesting they work together to suppress transposons and prevent tumorigenesis.

bioRxiv · Feb 6, 2023 · PreprintCancer Biology

The Mettl3 epitranscriptomic writer amplifies p53 stress responses

N. Raj, M. Wang, J. A. Seoane, N. A. Moonie, J. Demeter, A. M. Kaiser, A. M. Boutelle, A. S. Mulligan, C. Moffatt, C. Curtis, H. Chang, P. K. Jackson, L. D. Attardi

This paper identifies Mettl3 as a p53-interacting protein that stabilizes p53 and enhances its tumor suppressive functions through both protein stabilization and m6A modification of p53 pathway transcripts.

bioRxiv · Oct 21, 2021Cancer Biology