Author

P. Sicard

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2 Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Assay:imaging· 2 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

S. Richard· 3 shared A. Bonaud· 1 shared A. Carpinteiro· 1 shared A. Deney· 1 shared A. Jaccard· 1 shared A. Lampis· 1 shared A. Montagner· 1 shared A. Nicoletti· 1 shared

Activity by Year

2022-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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3 shared papersPhysiology

S. Richard

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

A mouse model of cardiac AL amyloidosis unveils mechanisms of tissue accumulation and toxicity of amyloid fibrils

G. Martinez-Rivas, M. V. Ayala, S. Bender, G. R. Codo, K. W. Swiderska, A. Lampis, L. Pedroza, M. Merdanovic, P. Sicard, E. Pinault, L. Richard, F. Lavatelli, D. Canetti, A. Rinsant, K. Sihem, , C. Sirac

This paper develops a mouse model of cardiac AL amyloidosis that reproduces human disease features, including amyloid deposits and cardiac dysfunction, and identifies a role for partial LC degradation in fibril formation.

Nature Communications · Mar 27, 2025Peer-reviewedPathology

Opposing Strain Directions on Adjacent Left Ventricular Segments Predict Fibrotic Remodeling after Acute Myocardial Infarction

T. Dubois-Mignon, V. Syvannarath, M. Para, S. Richard, P. Sicard, A. Nicoletti, G. Caligiuri, G. Franck

This study shows that opposing strain directions on adjacent left ventricular segments predict fibrotic remodeling after myocardial infarction, linking mechanical cues to transcriptional programs.

bioRxiv · Mar 20, 2023 · PreprintPhysiology

The autism-associated Meis2 gene is necessary for cardiac baroreflex regulation in mice

J. Roussel, R. LARCHER, P. SICARD, P. Bideaux, S. RICHARD, J. Thireau, F. MARMIGERE

This study shows that Meis2 gene inactivation in mouse peripheral sensory neurons impairs cardiac baroreflex regulation, linking ASD-related sensory neuron dysfunction to cardiovascular autonomic imbalance.

bioRxiv · Sep 14, 2022Physiology

Crest maturation at the cardiomyocyte surface contributes to a new late postnatal development stage that controls the diastolic function of the adult heart

c. karsenty, c. frugier, G. GENET, M.-H. SEGUELAS, P. Alzieu, O. Cazorla, A. Montagner, Y. Blum, c. Dubroca, J. Maupoint, B. Tramunt, M. Cauquil, T. Sulpice, S. Richard, S. Arcucci, , C. Gales

This study identifies a late postnatal developmental stage in rodents where cardiomyocyte surface crests mature, regulating diastolic function through ephrin-B1-dependent mechanisms.

bioRxiv · Feb 11, 2022Cell Biology