Author

M. V. Ayala

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Research Focus:differential expression· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Organism:human· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

C. Sirac· 3 shared M. Cogne· 3 shared S. Bender· 3 shared A. Bonaud· 2 shared A. Jaccard· 2 shared C. Carrion· 2 shared F. Bridoux· 2 shared V. Javaugue· 2 shared

Activity by Year

2019-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 44 mutual co-author links

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3 shared papersImmunology

C. Sirac

Mutual co-authors in cluster

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3 papers

Recent·Most cited

New models to study plasma cells in mouse based on the restriction of IgJ expression to antibody secreting cells.

M. V. Ayala, A. Bonaud, S. Bender, J.-M. Lambert, F. Lechouane, C. Carrion, M. Cogne, V. Pascal, C. Sirac

The paper introduces two new mouse models, IgJGFP and IgJCreERT2, for studying plasma cells by leveraging IgJ expression as a marker for antibody-secreting cells.

bioRxiv · Aug 13, 2020 · PreprintImmunology

Full-length immunoglobulin high-throughput sequencing reveals specific novel mutational patterns in POEMS syndrome

S. Bender, V. Javaugue, A. Saintamand, M. V. Ayala, M. Alizadeh, M. Filloux, V. Pascal, N. Gachard, D. Lavergne, F. Auroy, M. Cogne, F. Bridoux, C. Sirac, A. Jaccard

This study uses high-throughput Ig repertoire sequencing to identify specific mutational patterns in POEMS syndrome, aiding in diagnosis and detection of low-abundance plasma cell clones.

bioRxiv · Oct 31, 2019Immunology

Glomerulosclerosis and kidney failure in a mouse model of monoclonal immunoglobulin light-chain deposition disease

S. Bender, M. V. Ayala, A. Bonaud, V. Javaugue, C. Carrion, C. Oblet, A. Rinsant, N. Quellard, S. Kaaki, Z. Oruc, F. Boyer, A. Paquet, N. Pons, B. Herve, M. O. Ashi, , C. Sirac

This study develops a mouse model of light chain deposition disease, showing that pathogenic light chains cause glomerulosclerosis and kidney failure, and identifies potential therapeutic targets.

bioRxiv · May 2, 2019 · PreprintPathology