Author

Parisa Razaz

Broad Institute

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioinformatics· 1 Genomics· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1

Frequent Co-Authors

Alex R. HastieiD· 1 shared Alexei StortchevoiiD· 1 shared Anika GuptaiD· 1 shared April KimiD· 1 shared Ashok RagavendraniD· 1 shared Benjamin CurralliD· 1 shared Celine E. de Esch· 1 shared Dadi GaoiD· 1 shared

Activity by Year

2017-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperGenomics

Alex R. HastieORCID

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2 papers

Recent·Most cited

Tissue and cell-type specific molecular and functional signatures of 16p11.2 reciprocal genomic disorder across mouse brain and human neuronal models

Derek J.C. Tai, Parisa Razaz, Serkan Erdin, Dadi Gao, Jennifer P Wang, Xander Nuttle, Celine E. de Esch, Ryan L. Collins, Benjamin Currall, Kathryn O’Keefe, Nicholas D. Burt, Rachita Yadav, Lily Wang, Kiana Mohajeri, Tatsiana Aneichyk, , James F. Gusella

This study identifies molecular and functional changes in mouse and human models of 16p11.2 reciprocal genomic disorder, revealing context-specific disruptions in energy metabolism, RNA processing, and neuronal development.

The American Journal of Human Genetics · Sep 23, 2022Peer-reviewedGenomics

Detecting cancer vulnerabilities through gene networks under purifying selection in 4,700 cancer genomes

Anika Gupta, Heiko Horn, Parisa Razaz, April Kim, Michael S. Lawrence, Gad A. Getz, Kasper Lage

The paper identifies 685 putative cancer-related genes by analyzing mutation depletion in protein interaction networks across 4,700 cancer genomes.

bioRxiv · Nov 21, 2017 · PreprintBioinformatics