Author

Philipp G. Sämann

Max Planck Institute of Psychiatry

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Genetics· 1 Systems Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Alina TontschiD· 1 shared Angelika ErhardtiD· 1 shared Annalisa MarsicoiD· 1 shared BeCOME study group· 1 shared Elisabeth B. BinderiD· 1 shared Elisabeth Binder· 1 shared Gertrud EcksteiniD· 1 shared Ghalia RehawiiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperGenetics

Alina TontschORCID

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2 papers

Recent·Most cited

Integrative Gene and Isoform Co-expression Networks Reveal Regulatory Rewiring in Stress-related Psychiatric Disorders

Ghalia Rehawi, Jonas Hagenberg, BeCOME study group, OPTIMA study group, Philipp G. Sämann, Lambert Moyon, Elisabeth Binder, Markus List, Annalisa Marsico, Janine Arloth

The paper constructs co-expression networks using gene and isoform data from individuals with and without stress-related psychiatric disorders, revealing distinct network structures and regulatory rewiring.

iScience · Aug 13, 2025Peer-reviewedSystems Biology

Huntingtin CAG repeat size variations outside the Huntington's disease complex: associations with depression and anxiety phenotypes and basal ganglia structure

Magdalena Vater, Nicolas Rost, Gertrud Eckstein, Susann Sauer, Alina Tontsch, Angelika Erhardt, Susanne Lucae, Tanja Maria Brückl, Thomas Klopstock, Philipp G. Sämann, Elisabeth B. Binder

This study investigates the association between HTT CAG repeat size variations and psychiatric phenotypes and basal ganglia structure in non-HD ranges, finding age-dependent effects on anxiety and depression risk.

bioRxiv · May 22, 2024 · PreprintGenetics