Author

Gertrud Eckstein

Helmholtz MunichORCID 0000-0002-4755-230XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 2 Approach:statistical model· 2 Research Focus:differential expression· 1 Research Focus:GWAS / association mapping· 1 Assay:GWAS summary statistics· 1

Frequent Co-Authors

Alina TontschiD· 1 shared Angelika ErhardtiD· 1 shared Bernard D. KeavneyiD· 1 shared Bertram Müller‐MyhsokiD· 1 shared Claudia Abou‐Ajram· 1 shared Elda DziliciD· 1 shared Elisa Mastantuono· 1 shared Elisabeth B. BinderiD· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperGenetics

Alina TontschORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

GeneticsbioRxivPreprintMay 22, 2024

Huntingtin CAG repeat size variations outside the Huntington's disease complex: associations with depression and anxiety phenotypes and basal ganglia structure

Magdalena Vater, Nicolas Rost, Gertrud Eckstein…·Elisabeth B. Binder

This study investigates the association between HTT CAG repeat size variations and psychiatric phenotypes and basal ganglia structure in non-HD ranges, finding age-dependent effects on anxiety and depression risk.

1 citation

humanstatistical model

𝕏 PostBluesky

GenomicsJournal of Clinical InvestigationPeer-reviewedNov 17, 2020

Genome-wide association study in European patients with congenital heart disease identifies risk loci for transposition of the great arteries and anomalies of the thoracic arteries and veins and expression of discovered candidate genes in the developing heart

Harald Lahm, Meiwen Jia, Martina Dreßen…·Markus Krane

This study identifies genetic risk loci for congenital heart disease subtypes and shows candidate genes play essential roles in heart development.

112 citationstop 4%

humanGWAS summary statistics

𝕏 PostBluesky