Author

Preston R. Steele

University of Rhode Island

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Neuroscience· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:electronic health records· 3 Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Organism:rat· 2 Organism:mouse· 1

Frequent Co-Authors

Katharina A. QuinlaniD· 3 shared Alexander DrobyshevskyiD· 2 shared Clarissa F. CavarsaniD· 2 shared Emily J. ReedichiD· 2 shared Landon T. GenryiD· 2 shared Lisa Dowaliby· 2 shared Marin ManueliD· 2 shared Natallia KatenkaiD· 2 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersNeuroscience

Katharina A. QuinlanORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Spinal motoneurons respond aberrantly to serotonin in a rabbit model of cerebral palsy

Emily J. Reedich, Landon T. Genry, Preston R. Steele, Elvia Mena Avila, Lisa Dowaliby, Alexander Drobyshevsky, Marin Manuel, Katharina A. Quinlan

This study shows that spinal motoneurons in a rabbit model of cerebral palsy respond abnormally to serotonin, leading to hyperexcitability and spasticity.

The Journal of Physiology · Aug 16, 2023Peer-reviewedNeuroscience

Inhibitory interneurons show early dysfunction in a SOD1 mouse model of amyotrophic lateral sclerosis

Clarissa F. Cavarsan, Preston R. Steele, Landon T. Genry, Emily J. Reedich, Lynn M. McCane, Kay J. LaPre, Alyssa Puritz, Marin Manuel, Natallia Katenka, Katharina A. Quinlan

This study shows inhibitory interneurons in SOD1 mice exhibit early excitability and morphology changes, suggesting they contribute to ALS pathogenesis before motor neuron damage.

The Journal of Physiology · Dec 14, 2022Peer-reviewedNeuroscience

Altered motoneuron properties contribute to motor deficits in a rabbit hypoxia ischemia model of cerebral palsy

Preston R. Steele, Clarissa F. Cavarsan, Lisa Dowaliby, Megan Westefeld, Natallia Katenka, Alexander Drobyshevsky, Monica A. Gorassini, Katharina A. Quinlan

This study investigates how altered spinal motoneuron properties contribute to motor deficits in a rabbit hypoxia-ischemia model of cerebral palsy, finding significant physiological differences between affected and unaffected groups.

Frontiers in Cellular Neuroscience · Mar 25, 2020Peer-reviewedNeuroscience