Author

R. D. Morin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

K. Dreval· 2 shared M. Alcaide· 2 shared N. Thomas· 2 shared S. E. Arthur· 2 shared A. Mottok· 1 shared A. Telenius· 1 shared B. Grande· 1 shared C. K. Rushton· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersCancer Biology

K. Dreval

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2 papers

Recent·Most cited

Shared and distinct genetic features in human and canine B-cell lymphomas

K. M. Coyle, T. Hillman, M. Cheung, B. Grande, K. Bushell, S. E. Arthur, M. Alcaide, N. Thomas, K. Dreval, S. Wong, K. Campbell, R. D. Morin

This study compares genetic mutations in canine and human B-cell lymphomas, identifying shared and distinct features that may affect their biology and therapeutic development.

bioRxiv · Oct 14, 2021Cancer Biology

Non-coding NFKBIZ 3' UTR mutations promote cell growth and resistance to targeted therapies in diffuse large B-cell lymphoma

S. E. Arthur, J. Gao, S. Healy, C. K. Rushton, N. Thomas, L. K. Hilton, K. Dreval, J. Tang, M. Alcaide, R. Cojocaru, A. Mottok, A. Telenius, P. Unrau, W. H. Wilson, L. M. Staudt, , R. D. Morin

This study identifies non-coding NFKBIZ 3' UTR mutations as drivers of DLBCL growth and resistance to NF-κB-targeted therapies, suggesting their potential as biomarkers.

bioRxiv · May 22, 2021 · PreprintCancer Biology