Author

R. Delrosario

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:mouse· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. Balmain· 2 shared N. Bayani· 2 shared Q. Tran· 2 shared D. Wu· 1 shared E. Kandyba· 1 shared E. N. Bergstrom· 1 shared I.-J. Kim· 1 shared J. Digiovanni· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersCancer Biology

A. Balmain

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2 papers

Recent·Most cited

The interplay between dormant mutated cells and tumor promotion by chronic tissue damage in determining cancer risk.

Y. R. Li, E. Kandyba, K. Halliwill, R. Delrosario, Q. Tran, N. Bayani, D. Wu, O. Mirzoeva, M. M. Reeves, M. Islam, L. Riva, E. N. Bergstrom, K. Achanta, J. Digiovanni, L. Alexandrov, A. Balmain

This study analyzes mouse skin tumors to determine how environmental factors like TPA, obesity, and wounding affect mutational burden and cancer risk, finding that tumor promoters, not mutation number, limit tumor growth.

bioRxiv · Jan 24, 2024 · PreprintCancer Biology

Regulation of KRAS4A/B splicing in cancer stem cells by the RBM39 splicing complex.

W.-C. Chen, M. D. To, P. M. K. Westcott, R. Delrosario, I.-J. Kim, M. Phillips, Q. Tran, N. Bayani, A. Balmain

This study identifies KRAS4A and KRAS4B splice isoforms with distinct expression patterns in cancer stem cells and shows that targeting KRAS4A splicing is a potential therapeutic strategy in KRAS mutant tumors.

bioRxiv · May 22, 2019 · PreprintCancer Biology