Author

R. H. Crosbie

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:cell-cell communication· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. B. Willis· 1 shared A. D. Pyle· 1 shared A. H. Vo· 1 shared A. M. Long· 1 shared A. Maity· 1 shared A. R. Demonbreun· 1 shared A. Z. Stieg· 1 shared B. G. Novitch· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCell Biology

A. B. Willis

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Author profile →

2 papers

Recent·Most cited

The super-healing MRL strain promotes muscle growth in muscular dystrophy through a regenerative extracellular matrix

J. G. O'Brien, A. B. Willis, A. M. Long, J. Kwon, G. Lee, F. Li, P. G. Page, A. H. Vo, M. Hadhazy, R. H. Crosbie, A. R. Demonbreun, E. M. McNally

This paper shows that the MRL mouse strain's extracellular matrix promotes muscle regeneration in muscular dystrophy by reducing fibrosis and enhancing myoblast growth.

bioRxiv · Jun 29, 2023Cell Biology

Myoscaffolds reveal laminin scarring is detrimental for stem cell function while sarcospan induces compensatory fibrosis

K. M. Stearns-Reider, M. R. Hicks, K. G. Hammond, J. C. Reynolds, A. Maity, Y. Z. Kurmangaliyev, J. W. Chin, A. Z. Stieg, N. A. Geisse, S. Hohlbauch, S. Kaemmer, L. R. Schmitt, T. T. Pham, K. Yamauchi, B. G. Novitch, , R. H. Crosbie

The paper develops a myoscaffold platform to study fibrotic ECM effects on stem cell function, finding that laminin scarring impairs SMPCs while sarcospan induces compensatory fibrosis.

bioRxiv · Jul 7, 2022 · PreprintCell Biology