Author

J. C. Reynolds

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

A. D. Pyle· 1 shared A. Maity· 1 shared A. Z. Stieg· 1 shared B. A. Benayoun· 1 shared B. G. Novitch· 1 shared C. J. Mitchell· 1 shared C. Lee· 1 shared D. Cameron-Smith· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCell Biology

A. D. Pyle

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Author profile →

2 papers

Recent·Most cited

Myoscaffolds reveal laminin scarring is detrimental for stem cell function while sarcospan induces compensatory fibrosis

K. M. Stearns-Reider, M. R. Hicks, K. G. Hammond, J. C. Reynolds, A. Maity, Y. Z. Kurmangaliyev, J. W. Chin, A. Z. Stieg, N. A. Geisse, S. Hohlbauch, S. Kaemmer, L. R. Schmitt, T. T. Pham, K. Yamauchi, B. G. Novitch, , R. H. Crosbie

The paper develops a myoscaffold platform to study fibrotic ECM effects on stem cell function, finding that laminin scarring impairs SMPCs while sarcospan induces compensatory fibrosis.

bioRxiv · Jul 7, 2022 · PreprintCell Biology

MOTS-c is an Exercise-Induced Mitochondrial-Encoded Regulator of Age-Dependent Physical Decline and Muscle Homeostasis

J. C. Reynolds, R. W. Lai, J. S. T. Woodhead, J. H. Joly, C. J. Mitchell, D. Cameron-Smith, R. Lu, P. Cohen, N. A. Graham, B. A. Benayoun, T. L. Merry, C. Lee

This study shows that MOTS-c, a mitochondrial-encoded peptide, improves physical performance and muscle homeostasis across ages in mice and humans, suggesting mitochondrial genes regulate aging.

bioRxiv · Dec 22, 2019Cell Biology