Author

R. Salomon

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Cell Biology· 1 Immunology· 1 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 4 Organism:human· 4 Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Research Focus:cell-cell communication· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. Viau· 2 shared E. Birgy· 2 shared E. Poree· 2 shared K. Billot· 2 shared S. Saunier· 2 shared A. Belot· 1 shared A. Benmerah· 1 shared A. Bouafia· 1 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

A. Viau

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Prostaglandin E1 as therapeutic molecule for Nephronophthisis and related ciliopathies

H. Garcia, A. Serafin, F. Silbermann, E. Poree, C. Mahaut, A. Viau, K. Billot, E. Birgy, M. Garfa-Traore, S. Roy, S. Cecarelli, M. Mehraz, P. C. Rodriguez, B. Deleglise, L. Furio, , S. Saunier

This study identifies PGE1 as a potential therapeutic for NPH-associated ciliopathies, showing it rescues ciliogenesis and improves kidney phenotypes in models.

bioRxiv · Jan 21, 2022Cell Biology

The renal inflammatory network of nephronophthisis

M. Quatredeniers, F. Bienaime, G. Ferri, P. Isnard, E. Poree, K. Billot, E. Birgy, S. Ceccarelli, F. Legendre, S. Braeg, T. Nguyen-Khoa, R. Salomon, M.-C. Gubler, E. W. Kuehn, S. Saunier, A. Viau

This study identifies a CCL2-independent inflammatory network in nephronophthisis, revealing distinct immune cell infiltration and cytokine signatures compared to autosomal dominant polycystic kidney disease.

bioRxiv · Jan 7, 2021Physiology

Single cell transcriptomics reveals involution mimicry during the specification of the basal breast cancer subtype

F. Valdes-Mora, R. Salomon, B. S. Gloss, A. M. Law, L. Castillo, K. Murphy, J. Venhuizen, A. Magenau, M. Papanicolau, L. Rodriguez de la Fuente, D. L. Roden, Y. Colino-Sanguino, Z. Kikhtyak, N. Farbehi, J. R. Conway, , D. Gallego-Ortega

The paper uses single-cell RNA sequencing to show that basal breast cancer cells mimic involution processes, involving interactions with the tumor microenvironment to promote progression.

bioRxiv · May 16, 2020Cancer Biology

Heterozygous RELA mutations cause early-onset systemic lupus erythematosus by hijacking the NF-κB pathway towards transcriptional activation of type-I Interferon genes

L. Barnabei, H. Lamrini, M. Castela, N. Jeremiah, M.-C. Stolzenberg, L. Chentout, S. Mirshavalad, A. Bouafia, A. Magerus, M. Moncan, S. Jacques, O. Pelle, V. Bondet, D. Duffy, M. Parisot, , F. Rieux-Laucat

This study identifies heterozygous RELA mutations in early-onset SLE patients, showing they drive type-I interferon gene expression through NF-κB pathway hijacking.

bioRxiv · Apr 27, 2020 · PreprintImmunology