Author

A. Viau

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 3 Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 5 Organism:mouse· 4 Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Organism:human· 3 Research Focus:host-pathogen & immune response· 2 Assay:proteomics· 2 Organism:zebrafish· 2

Frequent Co-Authors

S. Saunier· 4 shared E. Poree· 3 shared E. W. Kuehn· 3 shared A. Benmerah· 2 shared E. Birgy· 2 shared F. Bienaime· 2 shared F. Silbermann· 2 shared G. Ferri· 2 shared

Activity by Year

2019-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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4 shared papersPathology

S. Saunier

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Author profile →

5 papers

Recent·Most cited

The ANKS3/BICC1 protein complex is a master post-transcriptional regulator of NPHP1 ciliopathy gene transcripts

A. Mahuzier, G. Odye, V. Grampa, A. A. Bizet, A. Viau, R. Ryan, M. Mehraz, T. Blisnick, E. Clark, C. Henry, R. Piedagnel, F. Silbermann, G. Hayot, L. De grande, A. Kahn, , S. Saunier

The paper identifies ANKS3/BICC1 as a post-transcriptional regulator of NPHP1 transcripts, linking it to ciliopathy phenotypes through transcript decay and ciliary dysfunction.

bioRxiv · Apr 18, 2024 · PreprintPathology

Prostaglandin E1 as therapeutic molecule for Nephronophthisis and related ciliopathies

H. Garcia, A. Serafin, F. Silbermann, E. Poree, C. Mahaut, A. Viau, K. Billot, E. Birgy, M. Garfa-Traore, S. Roy, S. Cecarelli, M. Mehraz, P. C. Rodriguez, B. Deleglise, L. Furio, , S. Saunier

This study identifies PGE1 as a potential therapeutic for NPH-associated ciliopathies, showing it rescues ciliogenesis and improves kidney phenotypes in models.

bioRxiv · Jan 21, 2022Cell Biology

YAP restricts renal inflammation and mitigates kidney damage in nephronothisis related kidney disease.

G. Ferri, V. Goffette, E. Poree, S. Braeg, S. Carvalho, M. Quatredeniers, E. W. Kuehn, S. Saunier, A. Viau

This study investigates the role of YAP in NPH-like kidney disease and finds that YAP inhibition is not therapeutic, suggesting LKB1 and YAP are parallel regulators of a harmful pathway.

bioRxiv · Jan 17, 2022 · PreprintPathology

The renal inflammatory network of nephronophthisis

M. Quatredeniers, F. Bienaime, G. Ferri, P. Isnard, E. Poree, K. Billot, E. Birgy, S. Ceccarelli, F. Legendre, S. Braeg, T. Nguyen-Khoa, R. Salomon, M.-C. Gubler, E. W. Kuehn, S. Saunier, A. Viau

This study identifies a CCL2-independent inflammatory network in nephronophthisis, revealing distinct immune cell infiltration and cytokine signatures compared to autosomal dominant polycystic kidney disease.

bioRxiv · Jan 7, 2021Physiology

Tubular STAT3 limits renal inflammation in autosomal dominant polycystic kidney disease

A. Viau, M. Baziz, A. Aka, C. Nguyen, E. W. Kuehn, F. Terzi, F. Bienaime

This study shows that STAT3 limits renal inflammation in ADPKD by repressing inflammatory chemokines, despite not driving cyst growth.

bioRxiv · Dec 12, 2019Pathology