Author

R. Shirakawa

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Approach:experimental assay· 3 Research Focus:gene regulatory network inference· 2 Assay:proteomics· 2 Organism:bacteria (other)· 1 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

C. Kaito· 2 shared K. Furuta· 2 shared K. Ishikawa· 2 shared A. D. Sarca· 1 shared A. Kubo· 1 shared A. Takaori-Kondo· 1 shared D. Takano· 1 shared E. Stanford· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersMicrobiology

C. Kaito

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Protective effects of Mkl1/2 against lipodystrophy and muscle atrophy via PI3K/AKT-independent FoxO repression

A. Kubo, K. Hitachi, R. Shirakawa, T. Ogura

This study shows Mkl1/2 repress FoxO transcription independently of PI3K/AKT, protecting against lipodystrophy and muscle atrophy by maintaining adipose and muscle tissue.

bioRxiv · Aug 9, 2023 · PreprintCell Biology

Knockout of ribosomal protein RpmJ leads to zinc resistance in Escherichia coli

R. Shirakawa, K. Ishikawa, K. Furuta, C. Kaito

The paper identifies RpmJ knockout in E. coli as a zinc-resistant mutation, showing decreased intracellular zinc and altered gene expression related to iron-sulfur clusters.

bioRxiv · Oct 24, 2022 · PreprintMicrobiology

Knockout of ykcB, a putative glycosyltransferase, leads to vancomycin resistance in Bacillus subtilis

K. Ishikawa, R. Shirakawa, D. Takano, T. Kosaki, K. Furuta, C. Kaito

This study identifies ykcB knockout in Bacillus subtilis as a cause of vancomycin resistance, with associated changes in cell surface composition and increased pathogenicity.

bioRxiv · Aug 30, 2022 · PreprintMicrobiology

APOBEC3B reporter myeloma cell lines identify DNA damage response pathways leading to APOBEC3B expression

H. Yamazaki, K. Shirakawa, T. Matsumoto, Y. Kazuma, H. Matsui, Y. Horisawa, E. Stanford, A. D. Sarca, R. Shirakawa, K. Shindo, A. Takaori-Kondo

The paper develops APOBEC3B reporter myeloma cell lines to study DNA damage response pathways that regulate APOBEC3B expression, finding that conventional therapies increase its expression while targeted drugs do not.

bioRxiv · Sep 24, 2019Cancer Biology