Author

R. T. Wheeler

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:zebrafish· 2 Organism:mouse· 1

Frequent Co-Authors

A. K. Scherer· 1 shared A. M. Dagher· 1 shared B. A. Blair· 1 shared B. G. Seman· 1 shared D. Aquilar· 1 shared J. B. Kelley· 1 shared J. Park· 1 shared M. S. Lionakis· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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1 shared paperMicrobiology

A. K. Scherer

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3 papers

Recent·Most cited

IL-23 signaling prevents ferroptosis-driven renal immunopathology during candidiasis

N. Millet, N. V. Solis, D. Aquilar, M. S. Lionakis, R. T. Wheeler, N. Jendzjowsky, M. Swidergall

This study identifies IL-23 signaling as a protective mechanism against ferroptosis-driven renal immunopathology during disseminated candidiasis.

bioRxiv · Dec 19, 2021Immunology

Pseudomonas synergizes with fluconazole against Candida during treatment of polymicrobial infection

S. Hattab, A. M. Dagher, R. T. Wheeler

This study shows that Pseudomonas aeruginosa enhances fluconazole's effectiveness against Candida albicans in co-infection models, partly through iron piracy.

bioRxiv · Nov 15, 2021Microbiology

Redundant Trojan horse and endothelial-circulatory mechanisms for host-mediated spread of Candida albicans yeast

A. K. Scherer, B. A. Blair, J. Park, B. G. Seman, J. B. Kelley, R. T. Wheeler

This study identifies redundant mechanisms by which Candida albicans spreads in zebrafish, using immune cells or blood flow.

PLOS Pathogens · Aug 10, 2020Peer-reviewedMicrobiology