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Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.
10 papers
Recent·Most cited
D. Aguilar, F. Zhu, A. Millet, N. Millet, P. Germano, J. Pisegna, O. Akbari, T. A. Doherty, M. Swidergall, N. Jendzjowsky
This study shows sensory neurons regulate humoral immunity by releasing neuropeptides that influence B-cell responses and antibody production in bacterial infection and allergic asthma.
bioRxiv · Jan 4, 2024 · PreprintImmunology
Q. T. Phan, N. V. Solis, M. V. Cravener, M. Swidergall, J. Lin, M. Y. Huang, H. Liu, S. Singh, A. S. Ibrahim, M. Mazzone, A. P. Mitchell, S. G. Filler
This study identifies a multi-receptor complex involving c-Met, E-cadherin, and EGFR that C. albicans uses to invade oral epithelial cells, with Hyr1 and Als3 facilitating this process and dual inhibition of c-Met and EGFR reducing virulence.
bioRxiv · Feb 23, 2023 · PreprintMicrobiology
N. Millet, N. V. Solis, D. Aquilar, M. S. Lionakis, R. T. Wheeler, N. Jendzjowsky, M. Swidergall
This study identifies IL-23 signaling as a protective mechanism against ferroptosis-driven renal immunopathology during disseminated candidiasis.
bioRxiv · Dec 19, 2021Immunology
Q. T. Phan, N. V. Solis, J. Lin, M. Swidergall, S. Singh, H. Liu, D. Sheppard, A. Ibrahim, A. P. Mitchell, S. G. Filler
This paper identifies serum proteins that mediate endothelial cell invasion by non-C. albicans Candida species through bridging molecule-mediated endocytosis.
bioRxiv · Sep 18, 2021 · PreprintMicrobiology
Q. T. Phan, J. Lin, N. V. Solis, M. Eng, M. Swidergall, F. Wang, S. LI, S. L. Gaffen, T.-F. Chou, S. G. Filler
This study identifies proteins interacting with EGFR during C. albicans infection and shows that gC1qR is essential for EGFR signaling and immune responses.
bioRxiv · May 25, 2021Microbiology
S. Soliman, C. Baldin, Y. Gu, S. Singh, T. Gebremariam, M. Swidergall, A. Alqarihi, E. Youssef, S. Alkhazraji, A. Pikoulas, C. Perske, V. Venkataramani, A. Rich, V. M. Bruno, J. C. Dunning Hotopp, , A. S. Ibrahim
This paper identifies mucoricin, a ricin-like toxin produced by Mucorales fungi, critical for mucormycosis pathogenesis and a potential therapeutic target.
bioRxiv · Jan 13, 2021Microbiology
M. Yang, N. Solis, M. Marshall, R. A. Garleb, T. Zhou, D. Wang, M. Swidergall, E. Pearlman, S. Filler, H. Liu
This study shows that Eng1 and Ywp1 in C. albicans reduce Dectin-1 binding by masking β-glucans, thereby decreasing immune recognition and virulence.
bioRxiv · Sep 7, 2020Microbiology
M. Swidergall, N. V. Solis, N. Millet, M. Y. Huang, J. Lin, Q. Phan, M. D. Lazarus, Z. Wang, A. P. Mitchell, S. G. Filler
This study investigates how Candida albicans virulence factors Als3 and Ece1/Candidalysin interact with host receptors EphA2 and EGFR to induce inflammation during oropharyngeal candidiasis.
bioRxiv · May 7, 2020 · PreprintMicrobiology
A. Alqarihi, T. Gebremariam, Y. Gu, M. Swidergall, S. Alkhazraji, s. Soliman, V. Bruno, J. Edwards, S. Filler, P. Uppuluri, A. S. Ibrahim
This study identifies GRP78 and integrins as distinct host receptors for Rhizopus delemar invasion in nasal and alveolar epithelial cells, explaining differential mucormycosis susceptibility in DKA and hematologic malignancy patients.
bioRxiv · Apr 29, 2020Microbiology
M. Swidergall, N. V. Solis, S. G. Filler
This study identifies EphA2 as a pattern recognition receptor that detects fungal β-glucans on Candida albicans, triggering an inflammatory response in oral epithelial cells.
bioRxiv · May 8, 2017Microbiology