Author

R. Vankayalapati

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:host-pathogen & immune response· 3 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Organism:mouse· 2 Organism:human· 1 Assay:metabolomics· 1 Approach:statistical model· 1

Frequent Co-Authors

B. Samten· 2 shared D. Ansari· 2 shared G. Yi· 2 shared A. Labani-Motlagh· 1 shared A. Vankayalapati· 1 shared B. Yang· 1 shared B.-G. Jung· 1 shared D. Tripathi· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersMicrobiology

B. Samten

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

HIV-differentiated metabolite N-Acetyl-L-Alanine dysregulates human natural killer cell responses to Mycobacterium tuberculosis infection

B. Yang, T. Mukherjee, R. Radhakrishnan, P. Paidipally, D. Ansari, S. John, R. Vankayalapati, D. Tripathi, G. Yi

The paper identifies N-Acetyl-L-Alanine as a metabolite dysregulated by HIV that inhibits NK cell responses to Mycobacterium tuberculosis infection.

International Journal of Molecular Sciences · Apr 14, 2023Peer-reviewedImmunology

Bacteriophage therapy for the treatment of Mycobacterium tuberculosis infections in humanized mice

G. Yi, F. Yang, A. Labani-Motlagh, J. D. Moreira, D. Ansari, J. A. Bohorquez, S. Patel, F. Spagnolo, J. Florence, A. Vankayalapati, R. Vankayalapati, J. J. D. J. Dennehy, B. Samten

This study tests three bacteriophages against Mtb and finds that DS6A effectively kills Mtb in culture and infected macrophages, improving survival and reducing bacterial load in humanized mice.

bioRxiv · Jan 23, 2023Microbiology

Mycobacterium tuberculosis stimulates IL-1β production by macrophages in an ESAT-6 dependent manner with the involvement of serum amyloid A3

B.-G. Jung, R. Vankayalapati, B. Samten

This study shows that Mycobacterium tuberculosis stimulates IL-1β production in macrophages via ESAT-6, with serum amyloid A3 playing a required role.

bioRxiv · Apr 4, 2020Immunology