Author

R. Wysoczanski

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Bioengineering· 1 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:benchmarking· 1 Approach:deep learning· 1 Research Focus:dimensionality reduction· 1 Approach:experimental assay· 1 Assay:fluorescence / live-cell microscopy· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1

Frequent Co-Authors

A. C. Kendall· 1 shared A. Kolbeinsson· 1 shared A. Nicolaou· 1 shared A. W. Segal· 1 shared C. Bakal· 1 shared C. Dunsby· 1 shared D. J. Marks· 1 shared D. W. Gilroy· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperImmunology

A. C. Kendall

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Robust optical autofocus system utilizing neural networks trained for extended range and time-course and automated multiwell plate imaging including single molecule localization microscopy

J. Lightley, F. Görlitz, S. Kumar, R. Kalita, A. Kolbeinsson, E. Garcia, Y. Alexandrov, V. Bousgouni, R. Wysoczanski, P. Barnes, L. Donnelly, C. Bakal, C. Dunsby, M. A. A. Neil, S. Flaxman, P. M. W. French

The paper develops a neural network-based autofocus system for microscopy, improving accuracy and range for long-term and multiwell plate imaging.

bioRxiv · Mar 5, 2021Bioengineering

Ulcerative colitis is characterized by amplified acute inflammation with delayed resolution

R. Wysoczanski, A. C. Kendall, M. Motwani, R. Vega, F. Z. Rahman, S. McCartney, S. L. Bloom, A. Nicolaou, D. W. Gilroy, A. W. Segal, D. J. Marks

This study shows that ulcerative colitis patients have exaggerated and prolonged inflammatory responses to bacterial injection, which are mitigated by 5-aminosalicylates through pro-resolution lipid pathways.

bioRxiv · Dec 10, 2019 · PreprintImmunology